By F. Kor-Shach. Crown College.
But we will focus on getting rid of the cause of pain and healing the organs that are in pain so none of these methods are needed buy 600mg neurontin with amex. I am not talking about the pain of a broken bone purchase neurontin 400 mg otc, twisted ankle, bee sting or sunburn. I am not talking about the pain of a misaligned vertebra or stretch trauma in your leg muscles or arm muscles. All of these may have special names like rheumatoid arthritis, cluster headache, fibromyalgia, bursitis, tennis elbow and so on, but they are all the same phenomenon. Knowing that parasites and pollutants are the real culprits, let us get right down to the job of finding out which they are, where they come from, and how to get rid of them. Our cells try to keep their doorways tight-shut but, of course, they have to open to let food in, or hormones, or other life-signals. There is probably a specific electrical attraction between them and an exact physical fit. Your white blood cells are waiting for them, and will gobble them up in a grand feast. Step Three is to find the pollutants and identify them because this gives us a clue as to their source. An intriguing question will pop into your head as you search your organs for parasites and pollutants. Or do the bacteria come first, jamming open the doorways so the pollutants can enter? The only ones that get away are those that are stuck in doorways and ‘channels with pollutants in them! Fortunately we do not have to know exactly how parasites and pollution make us sick in order to get well. Searching For Bacteria In order to find which organs have the bacteria and which bacteria are present you will need to learn the new technology that makes all of this possible. This technology is a simple electronic circuit that is capable of trapping frequencies in such a way that you can hear them. If your pain returned how would you know if it was the same old bacteria or a new one? What You Will Find First we will study and cure pains of all kinds, starting with the toes and working our way up the body. The inside of your eyeball, the testi- cle, the interior of gallstones, the middle of a tooth abscess or the bowel contents are such places. Your zapper current, because it is high frequency, prefers to “go around” these items, rather than through them. But with repeated zapping, and herbal parasite treatment, you can decimate them, too, and stop reinfecting the rest of your body. The body produces quite a bit of uric acid and this should, of course, be excreted into the bladder by the kidneys. But if the kidneys are doing a poor job of this, levels in the body and blood stream rise. Hippuric acid is made in large amounts (about 1 gram/day) by the liver because it is a detoxification product. It makes no sense to con- sume benzoic acid, the common preservative, since this is what the body detoxifies into hippuric acid. If you cannot find your pulse just below your inner ankle your circulation is poor. Some people do not have pain although these acids and other deposits are present making their joints knobby and unbending. Toe deposits are made of the same crystals as kidney stones, which is why the Kidney Cleanse works for toe pain. But because these deposits are far away from the kidney, it takes longer than merely cleaning up kidneys. This will at the same time remove kidney crystals so that these are no longer a source of bacteria. Get teeth cavitations cleaned (cavitations are bone infec- tions in the jaw where a tooth was pulled; it never healed; see Dental Cleanup page 409). The effect lasts for days afterward showing it is not the dental anes- thetic that is responsible. This, too, can give immediate pain relief in the toes showing you they are a source for bacteria. Ordinary pH paper, as for fish tanks, is almost as accurate and will serve as well. Taking a calcium and magne- sium supplement at bedtime, drinking milk at bedtime, using baking soda at bedtime are all remedies to be tried.
Medical treatment generally is not prescribed for campylobacteriosis because recovery is usually rapid order 400 mg neurontin visa. Cholera quality neurontin 800mg, Legionellosis, salmonellosis, shigellosis, yersiniosis, are other bacterial diseases that can be transmitted through water. All bacteria in water are readily killed or inactivated with chlorine or other disinfectants. Viral Diseases or Viruses Hepatitis A is a common example of a viral disease that may be transmitted through water. The onset is usually abrupt with fever, malaise, loss of appetite, nausea, and abdominal discomfort, followed within a few days by jaundice. The disease varies in severity from a mild illness lasting one to two weeks, to a severely disabling disease lasting several months (rare). Hepatitis A outbreaks have been related to fecally contaminated water; food contaminated by infected food handlers, including sandwiches and salads that are not cooked or are handled after cooking; and raw or undercooked mollusks harvested from contaminated waters. Aseptic meningitis, polio, and viral gastroenteritis (Norwalk agent) are other viral diseases that can be transmitted through water. Most viruses in drinking water can be inactivated by chlorine or other disinfectants. Terrorism Recent investigations have shown proof the terrorist organizations have been able to reproduce most of these pathogens and have the technology and funding to attack our public water supply system. Even diseases that we have not seen in years are easily and readily available for a terrorist to backflow into our distribution system, or pour into a wellhead or clearwell. Most of the following information may be simple or instruction that you already know. History of Research By the last half of the 19th century, the microbial world was known to consist of protozoa, fungi, and bacteria, all visible with a light microscope. In the 1840s, the German scientist Jacob Henle suggested that there were infectious agents too small to be seen with a light microscope, but for the lack of direct proof, his hypothesis was not accepted. Although the French scientist Louis Pasteur was working to develop a vaccine for rabies in the 1880s, he did not understand the concept of a virus. During the last half of the 19th century, several key discoveries were made that set the stage for the discovery of viruses. Pasteur is usually credited for dispelling the notion of spontaneous generation and proving that organisms reproduce new organisms. The German scientist Robert Koch, a student of Jacob Henle, and the British surgeon Joseph Lister developed techniques for growing cultures of single organisms that allowed the assignment of specific bacteria to specific diseases. Because Mayer was unable to isolate a bacterium or fungus from the tobacco leaf extracts, he considered the idea that tobacco mosaic disease might be caused by a soluble agent, but he concluded incorrectly that a new type of bacteria was likely to be the cause. The Russian scientist Dimitri Ivanofsky extended Mayer’s observation and reported in 1892 that the tobacco mosaic agent was small enough to pass through a porcelain filter known to block the passage of bacteria. But Ivanofsky, like Mayer, was bound by the dogma of his times and concluded in 1903 that the filter might be defective or that the disease agent was a toxin rather than a reproducing organism. Unaware of Ivanofsky’s results, the Dutch scientist Martinus Beijerinck, who collaborated with Mayer, repeated the filter experiment but extended this finding by demonstrating that the filtered material was not a toxin because it could grow and reproduce in the cells of the plant tissues. In his 1898 publication, Beijerinck referred to this new disease agent as a contagious living liquid—contagium vivum fluid—initiating a 20-year controversy over whether viruses were liquids or particles. The conclusion that viruses are particles came from several important observations. Because each hole, or plaque, developed from a single bacteriophage, this experiment provided the first method for counting infectious viruses (the plaque assay). In 1935 the American biochemist Wendell Meredith Stanley crystallized tobacco mosaic virus to demonstrate that viruses had regular shapes, and in 1939 tobacco mosaic virus was first visualized using the electron microscope. Frosch (both trained by Robert Koch) described foot-and-mouth disease virus as the first filterable agent of animals, and in 1900, the American bacteriologist Walter Reed and colleagues recognized yellow fever virus as the first human filterable agent. For several decades viruses were referred to as filterable agents, and gradually the term virus (Latin for “slimy liquid” or “poison”) was employed strictly for this new class of infectious agents. Through the 1940s and 1950s many critical discoveries were made about viruses through the study of bacteriophages because of the ease with which the bacteria they infect could be grown in the laboratory. Germ Theory of Disease History Louis Pasteur along with Robert Koch developed the germ theory of disease which states that "a specific disease is caused by a specific type of microorganism. Koch’s postulates not only proved the germ theory, but also gave a tremendous boost to the development of microbiology by stressing a laboratory culture and identification of microorganisms. Circumstances under which Koch’s postulates do not easily apply • Many healthy people carry pathogens but do not exhibit the symptoms of disease. These "carriers" may transmit the pathogens to others who then may become diseased. Example: viruses, chlamydia, rickettsias, and bacteria that cause leprosy and syphilis. Some of the fastidious organisms can now be grown in cultures of human or animal cells or in small animals. These secondary invaders or opportunists cause disease only when a person is ill or recovering from another disease.
Epidemic measures: Because of low infectivity and long incubation period purchase 100mg neurontin amex, epidemics of ﬁlariasis are almost unlikely neurontin 400 mg discount. The worm lodges in a pulmonary artery, where it may form the nidus of a thrombus; this can then lead to vascular occlusion, coagulation, necrosis and ﬁbrosis. A ﬁbrotic nodule, 1–3 cm in diameter, which most commonly is asymptomatic, is recognizable by X-ray as a “coin lesion. The worms develop in or migrate to the conjunctivae and the subcutaneous tissues of the scrotum, breasts, arms and legs, but microﬁlaraemia is rare. Diagnosis is usually made by the ﬁnding of worms in tissue sections of surgically excised lesions. These include Onchocerca volvulus and Loa loa, which cause onchocer- ciasis and loiasis, respectively (see under each disease listing). Infec- tion is usually asymptomatic, but eye infection from immature stages has been reported. Infection is generally asymptomatic but may be associated with allergic manifestations such as arthralgia, pruritus, headaches and lymphadenopathy. This chapter deals speciﬁcally with toxin-related foodborne illnesses (with the exception of botulism). Foodborne illnesses associated with infection by speciﬁc agents are covered in chapters dealing with these agents. Foodborne disease outbreaks are recognized by the occurrence of illness within a variable but usually short time period (a few hours to a few weeks) after a meal, among individuals who have consumed foods in common. Prompt and thorough laboratory evaluation of cases and implicated foods is essential. Single cases of foodborne disease are difﬁcult to identify unless, as in botulism, there is a distinctive clinical syndrome. Foodborne disease may be one of the most common causes of acute illness; many cases and outbreaks are unrecognized and unreported. Prevention and control of these diseases, regardless of speciﬁc cause, are based on the same principles: avoiding food contamination, destroying or denaturing contaminants, preventing further spread or multiplication of these contaminants. Speciﬁc problems and appropriate modes of interven- tion may vary from one country to another and depend on environmental, economic, political, technological and sociocultural factors. Ultimately, prevention depends on educating food handlers about proper practices in cooking and storage of food and personal hygiene. Identiﬁcation—An intoxication (not an infection) of abrupt and sometimes violent onset, with severe nausea, cramps, vomiting and prostration, often accompanied by diarrhea and sometimes with subnor- mal temperature and lowered blood pressure. Deaths are rare; illness commonly lasts only a day or two, but can take longer in severe cases; in rare cases, the intensity of symptoms may require hospitalization and surgical exploration. Differential diagnosis includes other recognized forms of food poisoning as well as chemical poisons. In the outbreak setting, recovery of large numbers of staphylococci (105 organisms or more/gram of food) on routine culture media, or detection of enterotoxin from an epidemiologically implicated food item conﬁrms the diagnosis. Absence of staphylococci on culture from heated food does not rule out the diagnosis; a Gram stain of the food may disclose the organisms that have been heat killed. It may be possible to identify enterotoxin or thermonuclease in the food in the absence of viable organisms. Isolation of organisms of the same phage type from stools or vomitus of 2 or more ill persons conﬁrms the diagnosis. Recovery of large numbers of enterotoxin- producing staphylococci from stool or vomitus from a single person supports the diagnosis. Phage typing and enterotoxin tests may help epidemiological investigations but are not routinely available or indicated; in outbreak settings, pulsed ﬁeld gel electrophoresis may be more useful in subtyping strains. Toxic agent—Several enterotoxins of Staphylococcus aureus, sta- ble at boiling temperature, even by thermal process. Staphylococci multiply in food and produce the toxins at levels of water activity too low for the growth of many competing bacteria. Occurrence—Widespread and relatively frequent; one of the prin- cipal acute food intoxications worldwide. Reservoir—Humans in most instances; occasionally cows with infected udders, as well as dogs and fowl. Toxin has also developed in inadequately cured ham and salami, and in unprocessed or inadequately processed cheese. When these foods remain at room tem- perature for several hours before being eaten, toxin-producing staphylo- cocci multiply and elaborate the heat-stable toxin. Organisms may be of human origin from purulent discharges of an infected ﬁnger or eye, abscesses, acneiform facial eruptions, nasopharyn- geal secretions or apparently normal skin; or of bovine origin, such as contaminated milk or milk products, especially cheese. Incubation period—Interval between eating food and onset of symptoms is 30 minutes to 8 hours, usually 2–4 hours. Preventive measures: 1) Educate food handlers about: (a) strict food hygiene, sani- tation and cleanliness of kitchens, proper temperature control, handwashing, cleaning of ﬁngernails; (b) the dan- ger of working with exposed skin, nose or eye infections and uncovered wounds. If they are to be stored for more than 2 hours, keep perishable foods hot (above 60°C/140°F) or cold (below 7°C/45°F; best is below 4°C/39°F) in shallow con- tainers and covered.
In places where the eczema is active cheap neurontin 400mg with visa, the skin is red from the increased blood supply and swollen because of the oedema buy generic neurontin 600mg. Symptoms of skin disorder Skin disease causes pruritus (itching), pain, soreness and discomfort, difficulty with movements of the hands and fingers, and cosmetic disability. Any skin abnormality can give rise to irritation, but some, such as scabies, seem particularly able to cause severe pruritus. Most scabies patients complain that their symptom of itch is much worse at night when they get warm, but this is probably not specific to this disorder. Itching in atopic der- matitis, senile pruritus and senile xerosis is made worse by repeated bathing and vigorous towelling afterwards, as well as by central heating and air conditioning with low relative humidity. If pruritus is made worse by aspirin or food additives such as tartrazine, sodium benzoate or the cinnamates, it is quite likely that 20 Symptoms of skin disorder urticaria is to blame. Persistent severe pruritus can be the most disabling and dis- tressing symptom, which is quite difficult to relieve. Scratching provides partial and transient relief from the symptom and it is fruitless to request that the patient stop scratching. Scratching itself causes damage to the skin surface, which is visi- ble as scratch marks (excoriations). In some patients, the repeated scratching and rubbing cause lichenification and in others prurigo papules occur. Uncommonly, the underlying disorder occurs at the site of the injury from the scratch. This phenomenon is found in patients with psoriasis and lichen planus and is known as the isomorphic response or the Koebner phenomenon. The notable exception to this is shin- gles (herpes zoster), which may cause pain and distorted sensations in the nerve root involved (see page 52). The pain may be present before the skin lesions appear, while they are there and, occasionally, afterwards. Pain and tenderness are characteristic of acutely inflamed lesions such as boils, acne cysts, cellulitis and erythema nodosum (see page 77). Most skin tumours are not painful, at least until they enlarge and infiltrate nerves. However, there are some uncommon benign tumours that cause pain, including the benign vascular tumour known as the glo- mus tumour and the benign tumour of plain muscle known as the leiomyoma. Chronic ulcers are often ‘sore’ and cause a variety of other discomforts, but they are not often the cause of severe pain. Painful fissures in the palms and soles develop in patches of eczema and psoriasis due to the inelastic, abnormal, horny layer in these conditions. For reasons that are not altogether clear, there is a primitive fear of diseased skin, which even amounts to feelings of disgust and revulsion. The idea of touching skin that is scal- ing or exudative seems inherently distasteful and it is something that one tries to avoid. These attitudes appear universal and inherent, and it is difficult to prevent them. It is little use pointing out that there is no rational basis for them, and all that can be hoped for is that a mixture of comprehension, compassion and common sense eventually supplants the primitive revulsion felt by all. It has been suggested that the origins of the inherent fear described above are the contagious nature of lep- rosy and the infestations of scabies and lice. Regardless of the origins, it is only too abundantly evident that individuals with obvious skin disease do not do well where the choice of others is concerned. They suffer more unemployment overall, but in addition 21 Signs and symptoms of skin disease Figure 2. Young patients with acne have particular problems because the disease is only too visible, as it usually affects the face. Psoriasis quite often affects the hands, nails and scalp margin, also causing difficulty for those whose occupations put them into contact with the public. Numerous other skin disorders put the affected individual at an economic and social disadvantage. Vascular birthmarks and large neurofibromata are disfiguring and tend to isolate the bearers. Chronic inflammatory facial disorders such as rosacea and discoid lupus erythematosus also cause problems (Figs 2. To summarize this point, individuals with visibly disordered skin are disabled because of society’s inherent avoidance reaction. One other aspect of this same problem is the sufferers’ own perception of the impact they are making on all with whom they come in contact. In most subjects who have persistent, ‘unsightly’ skin problems, the affected individuals become depressed and isolated.
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